SIVA1
SIVA1 (SIVA1 apoptosis inducing factor) هوَ بروتين يُشَفر بواسطة جين SIVA1 في الإنسان.[1][2]
الوظيفة
المراجع
- Prasad KV، Ao Z، Yoon Y، Wu MX، Rizk M، Jacquot S، Schlossman SF (يوليو 1997). "CD27, a member of the tumor necrosis factor receptor family, induces apoptosis and binds to Siva, a proapoptotic protein". Proc Natl Acad Sci U S A. ج. 94 ع. 12: 6346–51. DOI:10.1073/pnas.94.12.6346. PMC:21052. PMID:9177220.
- "Entrez Gene: SIVA1 SIVA1, apoptosis-inducing factor". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Maruyama K، Sugano S (1994). "Oligo-capping: a simple method to replace the cap structure of eukaryotic mRNAs with oligoribonucleotides". Gene. ج. 138 ع. 1–2: 171–4. DOI:10.1016/0378-1119(94)90802-8. PMID:8125298.
- Suzuki Y، Yoshitomo-Nakagawa K، Maruyama K، وآخرون (1997). "Construction and characterization of a full length-enriched and a 5'-end-enriched cDNA library". Gene. ج. 200 ع. 1–2: 149–56. DOI:10.1016/S0378-1119(97)00411-3. PMID:9373149.
- Henke A، Launhardt H، Klement K، وآخرون (2000). "Apoptosis in coxsackievirus B3-caused diseases: interaction between the capsid protein VP2 and the proapoptotic protein siva". J. Virol. ج. 74 ع. 9: 4284–90. DOI:10.1128/JVI.74.9.4284-4290.2000. PMC:111945. PMID:10756043.
- Cao C، Ren X، Kharbanda S، وآخرون (2001). "The ARG tyrosine kinase interacts with Siva-1 in the apoptotic response to oxidative stress". J. Biol. Chem. ج. 276 ع. 15: 11465–8. DOI:10.1074/jbc.C100050200. PMC:3476318. PMID:11278261.
- Henke A، Nestler M، Strunze S، وآخرون (2001). "The apoptotic capability of coxsackievirus B3 is influenced by the efficient interaction between the capsid protein VP2 and the proapoptotic host protein Siva". Virology. ج. 289 ع. 1: 15–22. DOI:10.1006/viro.2001.1082. PMID:11601913.
- Held-Feindt J، Mentlein R (2002). "CD70/CD27 ligand, a member of the TNF family, is expressed in human brain tumors". Int. J. Cancer. ج. 98 ع. 3: 352–6. DOI:10.1002/ijc.10207. PMID:11920585.
- Xue L، Chu F، Cheng Y، وآخرون (2002). "Siva-1 binds to and inhibits BCL-X(L)-mediated protection against UV radiation-induced apoptosis". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 10: 6925–30. DOI:10.1073/pnas.102182299. PMC:124505. PMID:12011449.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Spinicelli S، Nocentini G، Ronchetti S، وآخرون (2003). "GITR interacts with the pro-apoptotic protein Siva and induces apoptosis". Cell Death Differ. ج. 9 ع. 12: 1382–4. DOI:10.1038/sj.cdd.4401140. PMID:12478477.
- Chu F، Borthakur A، Sun X، وآخرون (2004). "The Siva-1 putative amphipathic helical region (SAH) is sufficient to bind to BCL-XL and sensitize cells to UV radiation induced apoptosis". Apoptosis. ج. 9 ع. 1: 83–95. DOI:10.1023/B:APPT.0000012125.01799.4c. PMID:14739602.
- Py B، Slomianny C، Auberger P، وآخرون (2004). "Siva-1 and an alternative splice form lacking the death domain, Siva-2, similarly induce apoptosis in T lymphocytes via a caspase-dependent mitochondrial pathway". J. Immunol. ج. 172 ع. 7: 4008–17. DOI:10.4049/jimmunol.172.7.4008. PMID:15034012.
- Fortin A، MacLaurin JG، Arbour N، وآخرون (2004). "The proapoptotic gene SIVA is a direct transcriptional target for the tumor suppressors p53 and E2F1". J. Biol. Chem. ج. 279 ع. 27: 28706–14. DOI:10.1074/jbc.M400376200. PMID:15105421.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Chu F، Barkinge J، Hawkins S، وآخرون (2005). "Expression of Siva-1 protein or its putative amphipathic helical region enhances cisplatin-induced apoptosis in breast cancer cells: effect of elevated levels of BCL-2". Cancer Res. ج. 65 ع. 12: 5301–9. DOI:10.1158/0008-5472.CAN-04-3270. PMID:15958577.
- Gudi R، Barkinge J، Hawkins S، وآخرون (2006). "Siva-1 negatively regulates NF-kappaB activity: effect on T-cell receptor-mediated activation-induced cell death (AICD)". Oncogene. ج. 25 ع. 24: 3458–62. DOI:10.1038/sj.onc.1209381. PMID:16491128.
- Nestler M، Martin U، Hortschansky P، وآخرون (2006). "The zinc containing pro-apoptotic protein siva interacts with the peroxisomal membrane protein pmp22". Mol. Cell. Biochem. ج. 287 ع. 1–2: 147–55. DOI:10.1007/s11010-005-9082-6. PMID:16683188.
- Severino A، Abbruzzese C، Manente L، وآخرون (2007). "Human papillomavirus-16 E7 interacts with Siva-1 and modulates apoptosis in HaCaT human immortalized keratinocytes". J. Cell. Physiol. ج. 212 ع. 1: 118–25. DOI:10.1002/jcp.21011. PMID:17348035.
بوابة طب
بوابة الكيمياء الحيوية
بوابة علم الأحياء الخلوي والجزيئي
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.