SEMA5A
SEMA5A (Semaphorin 5A) هوَ بروتين يُشَفر بواسطة جين SEMA5A في الإنسان.[1][2][3]
الوظيفة
المراجع
- Simmons AD، Puschel AW، McPherson JD، Overhauser J، Lovett M (مارس 1998). "Molecular cloning and mapping of human semaphorin F from the Cri-du-chat candidate interval". Biochem Biophys Res Commun. ج. 242 ع. 3: 685–91. DOI:10.1006/bbrc.1997.8027. PMID:9464278.
- "Entrez Gene: SEMA5A sema domain, seven thrombospondin repeats (type 1 and type 1-like), transmembrane domain (TM) and short cytoplasmic domain, (semaphorin) 5A". مؤرشف من الأصل في 2010-12-05.
- Adams RH، Betz H، Puschel AW (فبراير 1997). "A novel class of murine semaphorins with homology to thrombospondin is differentially expressed during early embryogenesis". Mech Dev. ج. 57 ع. 1: 33–45. DOI:10.1016/0925-4773(96)00525-4. PMID:8817451.
قراءة متعمقة
- Bonaldo MF، Lennon G، Soares MB (1997). "Normalization and subtraction: two approaches to facilitate gene discovery". Genome Res. ج. 6 ع. 9: 791–806. DOI:10.1101/gr.6.9.791. PMID:8889548.
- Simmons AD، Overhauser J، Lovett M (1997). "Isolation of cDNAs from the Cri-du-chat critical region by direct screening of a chromosome 5-specific cDNA library". Genome Res. ج. 7 ع. 2: 118–27. DOI:10.1101/gr.7.2.118. PMID:9049630.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Artigiani S، Conrotto P، Fazzari P، وآخرون (2005). "Plexin-B3 is a functional receptor for semaphorin 5A". EMBO Rep. ج. 5 ع. 7: 710–4. DOI:10.1038/sj.embor.7400189. PMC:1299100. PMID:15218527.
- Beausoleil SA، Jedrychowski M، Schwartz D، وآخرون (2004). "Large-scale characterization of HeLa cell nuclear phosphoproteins". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 33: 12130–5. DOI:10.1073/pnas.0404720101. PMC:514446. PMID:15302935.
- Schmutz J، Martin J، Terry A، وآخرون (2004). "The DNA sequence and comparative analysis of human chromosome 5". Nature. ج. 431 ع. 7006: 268–74. DOI:10.1038/nature02919. PMID:15372022.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Kimura K، Wakamatsu A، Suzuki Y، وآخرون (2006). "Diversification of transcriptional modulation: large-scale identification and characterization of putative alternative promoters of human genes". Genome Res. ج. 16 ع. 1: 55–65. DOI:10.1101/gr.4039406. PMC:1356129. PMID:16344560.
- Melin M، Carlsson B، Anckarsater H، وآخرون (2007). "Constitutional downregulation of SEMA5A expression in autism". Neuropsychobiology. ج. 54 ع. 1: 64–9. DOI:10.1159/000096040. PMC:2553518. PMID:17028446.
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة طب
- بوابة الكيمياء الحيوية
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