PLEKHA5
PLEKHA5 (Pleckstrin homology domain containing A5) هوَ بروتين يُشَفر بواسطة جين PLEKHA5 في الإنسان.[1][2][3]
الوظيفة
المراجع
- Nagase T، Kikuno R، Hattori A، Kondo Y، Okumura K، Ohara O (فبراير 2001). "Prediction of the coding sequences of unidentified human genes. XIX. The complete sequences of 100 new cDNA clones from brain which code for large proteins in vitro". DNA Res. ج. 7 ع. 6: 347–55. DOI:10.1093/dnares/7.6.347. PMID:11214970.
- "Entrez Gene: PLEKHA5 pleckstrin homology domain containing, family A member 5". مؤرشف من الأصل في 2010-12-05.
- Dowler S، Currie RA، Campbell DG، Deak M، Kular G، Downes CP، Alessi DR (يناير 2001). "Identification of pleckstrin-homology-domain-containing proteins with novel phosphoinositide-binding specificities". Biochem J. ج. 351 ع. Pt 1: 19–31. DOI:10.1042/0264-6021:3510019. PMC:1221362. PMID:11001876.
قراءة متعمقة
- Nakajima D، Okazaki N، Yamakawa H، وآخرون (2003). "Construction of expression-ready cDNA clones for KIAA genes: manual curation of 330 KIAA cDNA clones". DNA Res. ج. 9 ع. 3: 99–106. DOI:10.1093/dnares/9.3.99. PMID:12168954.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Lehner B، Sanderson CM (2004). "A protein interaction framework for human mRNA degradation". Genome Res. ج. 14 ع. 7: 1315–23. DOI:10.1101/gr.2122004. PMC:442147. PMID:15231747.
- Jin J، Smith FD، Stark C، وآخرون (2004). "Proteomic, functional, and domain-based analysis of in vivo 14-3-3 binding proteins involved in cytoskeletal regulation and cellular organization". Curr. Biol. ج. 14 ع. 16: 1436–50. DOI:10.1016/j.cub.2004.07.051. PMID:15324660.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Zhang Y، Wolf-Yadlin A، Ross PL، وآخرون (2005). "Time-resolved mass spectrometry of tyrosine phosphorylation sites in the epidermal growth factor receptor signaling network reveals dynamic modules". Mol. Cell. Proteomics. ج. 4 ع. 9: 1240–50. DOI:10.1074/mcp.M500089-MCP200. PMID:15951569.
- Lim J، Hao T، Shaw C، وآخرون (2006). "A protein-protein interaction network for human inherited ataxias and disorders of Purkinje cell degeneration". Cell. ج. 125 ع. 4: 801–14. DOI:10.1016/j.cell.2006.03.032. PMID:16713569.
- Beausoleil SA، Villén J، Gerber SA، وآخرون (2006). "A probability-based approach for high-throughput protein phosphorylation analysis and site localization". Nat. Biotechnol. ج. 24 ع. 10: 1285–92. DOI:10.1038/nbt1240. PMID:16964243.
- Olsen JV، Blagoev B، Gnad F، وآخرون (2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. ج. 127 ع. 3: 635–48. DOI:10.1016/j.cell.2006.09.026. PMID:17081983.
- بوابة الكيمياء الحيوية
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة طب
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