NFYB
NFYB (Nuclear transcription factor Y subunit beta) هوَ بروتين يُشَفر بواسطة جين NFYB في الإنسان.[1][2]
الوظيفة
المراجع
- Maity SN، de Crombrugghe B (يونيو 1998). "Role of the CCAAT-binding protein CBF/NF-Y in transcription". Trends Biochem Sci. ج. 23 ع. 5: 174–8. DOI:10.1016/S0968-0004(98)01201-8. PMID:9612081.
- Li XY، Mattei MG، Zaleska-Rutczynska Z، Hooft van Huijsduijnen R، Figueroa F، Nadeau J، Benoist C، Mathis D (مارس 1992). "One subunit of the transcription factor NF-Y maps close to the major histocompatibility complex in murine and human chromosomes". Genomics. ج. 11 ع. 3: 630–4. DOI:10.1016/0888-7543(91)90070-U. PMID:1774067.
قراءة متعمقة
- Lloberas J، Soler C، Celada A (1998). "Repression mechanisms of the I-A beta gene of the major histocompatibility complex". Immunobiology. ج. 198 ع. 1–3: 249–63. DOI:10.1016/s0171-2985(97)80045-9. PMID:9442396.
- Mantovani R (1999). "The molecular biology of the CCAAT-binding factor NF-Y". Gene. ج. 239 ع. 1: 15–27. DOI:10.1016/S0378-1119(99)00368-6. PMID:10571030.
- Li XY، Mantovani R، Hooft van Huijsduijnen R، Andre I، Benoist C، Mathis D (1992). "Evolutionary variation of the CCAAT-binding transcription factor NF-Y". Nucleic Acids Res. ج. 20 ع. 5: 1087–91. DOI:10.1093/nar/20.5.1087. PMC:312095. PMID:1549471.
- Li XY، Hooft van Huijsduijnen R، Mantovani R، Benoist C، Mathis D (1992). "Intron-exon organization of the NF-Y genes. Tissue-specific splicing modifies an activation domain". J. Biol. Chem. ج. 267 ع. 13: 8984–90. PMID:1577736.
- Lloberas J، Maki RA، Celada A (1995). "Repression of major histocompatibility complex I-A beta gene expression by dbpA and dbpB (mYB-1) proteins". Mol. Cell. Biol. ج. 15 ع. 9: 5092–9. PMC:230756. PMID:7651426.
- Chang ZF، Liu CJ (1994). "Human thymidine kinase CCAAT-binding protein is NF-Y, whose A subunit expression is serum-dependent in human IMR-90 diploid fibroblasts". J. Biol. Chem. ج. 269 ع. 27: 17893–8. PMID:8027044.
- Mantovani R، Li XY، Pessara U، Hooft van Huisjduijnen R، Benoist C، Mathis D (1994). "Dominant negative analogs of NF-YA". J. Biol. Chem. ج. 269 ع. 32: 20340–6. PMID:8051128.
- Pise-Masison CA، Dittmer J، Clemens KE، Brady JN (1997). "Physical and functional interaction between the human T-cell lymphotropic virus type 1 Tax1 protein and the CCAAT binding protein NF-Y". Mol. Cell. Biol. ج. 17 ع. 3: 1236–43. DOI:10.1128/mcb.17.3.1236. PMC:231848. PMID:9032250.
- Bellorini M، Lee DK، Dantonel JC، Zemzoumi K، Roeder RG، Tora L، Mantovani R (1997). "CCAAT binding NF-Y-TBP interactions: NF-YB and NF-YC require short domains adjacent to their histone fold motifs for association with TBP basic residues". Nucleic Acids Res. ج. 25 ع. 11: 2174–81. DOI:10.1093/nar/25.11.2174. PMC:146709. PMID:9153318.
- Currie RA (1998). "Functional interaction between the DNA binding subunit trimerization domain of NF-Y and the high mobility group protein HMG-I(Y)". J. Biol. Chem. ج. 272 ع. 49: 30880–8. DOI:10.1074/jbc.272.49.30880. PMID:9388234.
- Currie RA (1998). "NF-Y is associated with the histone acetyltransferases GCN5 and P/CAF". J. Biol. Chem. ج. 273 ع. 3: 1430–4. DOI:10.1074/jbc.273.3.1430. PMID:9430679.
- Currie RA (1998). "Biochemical characterization of the NF-Y transcription factor complex during B lymphocyte development". J. Biol. Chem. ج. 273 ع. 29: 18220–9. DOI:10.1074/jbc.273.29.18220. PMID:9660784.
- Bemark M، Olsson H، Heinegård D، Leanderson T (1998). "Purification and characterization of a protein binding to the SP6 kappa promoter. A potential role for CArG-box binding factor-A in kappa transcription". J. Biol. Chem. ج. 273 ع. 30: 18881–90. DOI:10.1074/jbc.273.30.18881. PMID:9668064.
- Faniello MC، Bevilacqua MA، Condorelli G، de Crombrugghe B، Maity SN، Avvedimento VE، Cimino F، Costanzo F (1999). "The B subunit of the CAAT-binding factor NFY binds the central segment of the Co-activator p300". J. Biol. Chem. ج. 274 ع. 12: 7623–6. DOI:10.1074/jbc.274.12.7623. PMID:10075648.
- Hake SB، Masternak K، Kammerbauer C، Janzen C، Reith W، Steimle V (2000). "CIITA leucine-rich repeats control nuclear localization, in vivo recruitment to the major histocompatibility complex (MHC) class II enhanceosome, and MHC class II gene transactivation". Mol. Cell. Biol. ج. 20 ع. 20: 7716–25. DOI:10.1128/MCB.20.20.7716-7725.2000. PMC:86349. PMID:11003667.
- Liang F، Schaufele F، Gardner DG (2001). "Functional interaction of NF-Y and Sp1 is required for type a natriuretic peptide receptor gene transcription". J. Biol. Chem. ج. 276 ع. 2: 1516–22. DOI:10.1074/jbc.M006350200. PMID:11022037.
- Izumi H، Molander C، Penn LZ، Ishisaki A، Kohno K، Funa K (2001). "Mechanism for the transcriptional repression by c-Myc on PDGF beta-receptor". J. Cell Sci. ج. 114 ع. Pt 8: 1533–44. PMID:11282029.
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة طب
- بوابة الكيمياء الحيوية
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