L3MBTL
L3MBTL (L3MBTL1, histone methyl-lysine binding protein) هوَ بروتين يُشَفر بواسطة جين L3MBTL في الإنسان.[1][2][3]
الوظيفة
المراجع
- Koga H، Matsui S، Hirota T، Takebayashi S، Okumura K، Saya H (سبتمبر 1999). "A human homolog of Drosophila lethal(3)malignant brain tumor (l(3)mbt) protein associates with condensed mitotic chromosomes". Oncogene. ج. 18 ع. 26: 3799–809. DOI:10.1038/sj.onc.1202732. PMID:10445843.
- "Entrez Gene: L3MBTL l(3)mbt-like (Drosophila)". مؤرشف من الأصل في 2010-12-05.
- Trojer P، Li G، Sims RJ III، Vaquero A، Kalakonda N، Boccuni P، Lee D، Erdjument-Bromage H، Tempst P، Nimer SD، Wang YH، Reinberg D (يونيو 2007). "L3MBTL1, a histone-methylation-dependent chromatin lock". Cell. ج. 129 ع. 5: 915–28. DOI:10.1016/j.cell.2007.03.048. PMID:17540172.
قراءة متعمقة
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- MacGrogan D، Kalakonda N، Alvarez S، وآخرون (2004). "Structural integrity and expression of the L3MBTL gene in normal and malignant hematopoietic cells". Genes Chromosomes Cancer. ج. 41 ع. 3: 203–13. DOI:10.1002/gcc.20087. PMID:15334543.
- Li J، Bench AJ، Vassiliou GS، وآخرون (2004). "Imprinting of the human L3MBTL gene, a polycomb family member located in a region of chromosome 20 deleted in human myeloid malignancies". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 19: 7341–6. DOI:10.1073/pnas.0308195101. PMC:409920. PMID:15123827.
- Wang WK، Tereshko V، Boccuni P، وآخرون (2004). "Malignant brain tumor repeats: a three-leaved propeller architecture with ligand/peptide binding pockets". Structure. ج. 11 ع. 7: 775–89. DOI:10.1016/S0969-2126(03)00127-8. PMID:12842041.
- Boccuni P، MacGrogan D، Scandura JM، Nimer SD (2003). "The human L(3)MBT polycomb group protein is a transcriptional repressor and interacts physically and functionally with TEL (ETV6)". J. Biol. Chem. ج. 278 ع. 17: 15412–20. DOI:10.1074/jbc.M300592200. PMID:12588862.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Deloukas P، Matthews LH، Ashurst J، وآخرون (2002). "The DNA sequence and comparative analysis of human chromosome 20". Nature. ج. 414 ع. 6866: 865–71. DOI:10.1038/414865a. PMID:11780052.
- Fossey SC، Mychaleckyj JC، Pendleton JK، وآخرون (2001). "A high-resolution 6.0-megabase transcript map of the type 2 diabetes susceptibility region on human chromosome 20". Genomics. ج. 76 ع. 1–3: 45–57. DOI:10.1006/geno.2001.6584. PMID:11549316.
- Dias Neto E، Correa RG، Verjovski-Almeida S، وآخرون (2000). "Shotgun sequencing of the human transcriptome with ORF expressed sequence tags". Proc. Natl. Acad. Sci. U.S.A. ج. 97 ع. 7: 3491–6. DOI:10.1073/pnas.97.7.3491. PMC:16267. PMID:10737800.
- Ishikawa K، Nagase T، Suyama M، وآخرون (1998). "Prediction of the coding sequences of unidentified human genes. X. The complete sequences of 100 new cDNA clones from brain which can code for large proteins in vitro". DNA Res. ج. 5 ع. 3: 169–76. DOI:10.1093/dnares/5.3.169. PMID:9734811.
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة الكيمياء الحيوية
- بوابة طب
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