GAS1
GAS1 (Growth arrest specific 1) هوَ بروتين يُشَفر بواسطة جين GAS1 في الإنسان.[1][2]
الوظيفة
المراجع
- "Entrez Gene: GAS1 growth arrest-specific 1". مؤرشف من الأصل في 2010-12-05.
- Evdokiou A، Webb GC، Peters GB، Dobrovic A، O'Keefe DS، Forbes IJ، Cowled PA (مارس 1994). "Localization of the human growth arrest-specific gene (GAS1) to chromosome bands 9q21.3-q22, a region frequently deleted in myeloid malignancies". Genomics. ج. 18 ع. 3: 731–3. DOI:10.1016/S0888-7543(05)80388-X. PMID:8307588.
قراءة متعمقة
- Del Sal G، Ruaro ME، Philipson L، Schneider C (1992). "The growth arrest-specific gene, gas1, is involved in growth suppression". Cell. ج. 70 ع. 4: 595–607. DOI:10.1016/0092-8674(92)90429-G. PMID:1505026.
- Wicking C، Breen M، Negus K، وآخرون (1994). "The human growth-arrest-specific gene GAS1 maps outside the candidate region of the gene for nevoid basal cell carcinoma syndrome". Cytogenet. Cell Genet. ج. 68 ع. 1–2: 119–21. DOI:10.1159/000133904. PMID:7956349.
- Del Sal G، Collavin L، Ruaro ME، وآخرون (1994). "Structure, function, and chromosome mapping of the growth-suppressing human homologue of the murine gas1 gene". Proc. Natl. Acad. Sci. U.S.A. ج. 91 ع. 5: 1848–52. DOI:10.1073/pnas.91.5.1848. PMC:43261. PMID:8127893.
- Evdokiou A، Cowled PA (1998). "Tumor-suppressive activity of the growth arrest-specific gene GAS1 in human tumor cell lines". Int. J. Cancer. ج. 75 ع. 4: 568–77. DOI:10.1002/(SICI)1097-0215(19980209)75:4<568::AID-IJC13>3.0.CO;2-5. PMID:9466658.
- Stebel M، Vatta P، Ruaro ME، وآخرون (2000). "The growth suppressing gas1 product is a GPI-linked protein". FEBS Lett. ج. 481 ع. 2: 152–8. DOI:10.1016/S0014-5793(00)02004-4. PMID:10996315.
- Lee CS، Buttitta L، Fan CM (2001). "Evidence that the WNT-inducible growth arrest-specific gene 1 encodes an antagonist of sonic hedgehog signaling in the somite". Proc. Natl. Acad. Sci. U.S.A. ج. 98 ع. 20: 11347–52. DOI:10.1073/pnas.201418298. PMC:58732. PMID:11572986.
- Baladrón V، Ruiz-Hidalgo MJ، Bonvini E، وآخرون (2002). "The EGF-like homeotic protein dlk affects cell growth and interacts with growth-modulating molecules in the yeast two-hybrid system". Biochem. Biophys. Res. Commun. ج. 291 ع. 2: 193–204. DOI:10.1006/bbrc.2002.6431. PMID:11846389.
- Mellström B، Ceña V، Lamas M، وآخرون (2002). "Gas1 is induced during and participates in excitotoxic neuronal death". Mol. Cell. Neurosci. ج. 19 ع. 3: 417–29. DOI:10.1006/mcne.2001.1092. PMID:11906213.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Xu Y، Kulkosky J، Acheampong E، وآخرون (2004). "HIV-1-mediated apoptosis of neuronal cells: Proximal molecular mechanisms of HIV-1-induced encephalopathy". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 18: 7070–5. DOI:10.1073/pnas.0304859101. PMC:406467. PMID:15103018.
- Cabrera JR، Sanchez-Pulido L، Rojas AM، وآخرون (2006). "Gas1 is related to the glial cell-derived neurotrophic factor family receptors alpha and regulates Ret signaling". J. Biol. Chem. ج. 281 ع. 20: 14330–9. DOI:10.1074/jbc.M509572200. PMID:16551639.
بوابة الكيمياء الحيوية
بوابة علم الأحياء الخلوي والجزيئي
بوابة طب
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