CTCFL
CTCFL (CCCTC-binding factor like) هوَ بروتين يُشَفر بواسطة جين CTCFL في الإنسان.[1]
الوظيفة
المراجع
- "Entrez Gene: CTCFL CCCTC-binding factor (zinc finger protein)-like". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Klenova EM، Morse HC، Ohlsson R، Lobanenkov VV (2003). "The novel BORIS + CTCF gene family is uniquely involved in the epigenetics of normal biology and cancer". Semin. Cancer Biol. ج. 12 ع. 5: 399–414. DOI:10.1016/S1044-579X(02)00060-3. PMID:12191639.
- D'Arcy V، Pore N، Docquier F، وآخرون (2008). "BORIS, a paralogue of the transcription factor, CTCF, is aberrantly expressed in breast tumours". Br. J. Cancer. ج. 98 ع. 3: 571–9. DOI:10.1038/sj.bjc.6604181. PMC:2243163. PMID:18195709.
- Woloszynska-Read A، James SR، Link PA، وآخرون (2008). "DNA methylation-dependent regulation of BORIS/CTCFL expression in ovarian cancer". Cancer Immun. ج. 7: 21. PMC:2935752. PMID:18095639.
- Renaud S، Pugacheva EM، Delgado MD، وآخرون (2008). "Expression of the CTCF-paralogous cancer-testis gene, brother of the regulator of imprinted sites (BORIS), is regulated by three alternative promoters modulated by CpG methylation and by CTCF and p53 transcription factors". Nucleic Acids Res. ج. 35 ع. 21: 7372–88. DOI:10.1093/nar/gkm896. PMC:2175345. PMID:17962299.
- Kholmanskikh O، Loriot A، Brasseur F، وآخرون (2008). "Expression of BORIS in melanoma: lack of association with MAGE-A1 activation". Int. J. Cancer. ج. 122 ع. 4: 777–84. DOI:10.1002/ijc.23140. PMID:17957795.
- Risinger JI، Chandramouli GV، Maxwell GL، وآخرون (2007). "Global expression analysis of cancer/testis genes in uterine cancers reveals a high incidence of BORIS expression". Clin. Cancer Res. ج. 13 ع. 6: 1713–9. DOI:10.1158/1078-0432.CCR-05-2569. PMID:17363524.
- D'Arcy V، Abdullaev ZK، Pore N، وآخرون (2006). "The potential of BORIS detected in the leukocytes of breast cancer patients as an early marker of tumorigenesis". Clin. Cancer Res. ج. 12 ع. 20 Pt 1: 5978–86. DOI:10.1158/1078-0432.CCR-05-2731. PMID:17062669.
- Hoffmann MJ، Müller M، Engers R، Schulz WA (2007). "Epigenetic control of CTCFL/BORIS and OCT4 expression in urogenital malignancies". Biochem. Pharmacol. ج. 72 ع. 11: 1577–88. DOI:10.1016/j.bcp.2006.06.020. PMID:16854382.
- Hong JA، Kang Y، Abdullaev Z، وآخرون (2005). "Reciprocal binding of CTCF and BORIS to the NY-ESO-1 promoter coincides with derepression of this cancer-testis gene in lung cancer cells". Cancer Res. ج. 65 ع. 17: 7763–74. DOI:10.1158/0008-5472.CAN-05-0823. PMID:16140944.
- Rodriguez M، Yu X، Chen J، Songyang Z (2004). "Phosphopeptide binding specificities of BRCA1 COOH-terminal (BRCT) domains". J. Biol. Chem. ج. 278 ع. 52: 52914–8. DOI:10.1074/jbc.C300407200. PMID:14578343.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Loukinov DI، Pugacheva E، Vatolin S، وآخرون (2002). "BORIS, a novel male germ-line-specific protein associated with epigenetic reprogramming events, shares the same 11-zinc-finger domain with CTCF, the insulator protein involved in reading imprinting marks in the soma". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 10: 6806–11. DOI:10.1073/pnas.092123699. PMC:124484. PMID:12011441.
- Deloukas P، Matthews LH، Ashurst J، وآخرون (2002). "The DNA sequence and comparative analysis of human chromosome 20". Nature. ج. 414 ع. 6866: 865–71. DOI:10.1038/414865a. PMID:11780052.
- بوابة الكيمياء الحيوية
- بوابة طب
- بوابة علم الأحياء الخلوي والجزيئي
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