CLDN5
CLDN5 (Claudin 5) هوَ بروتين يُشَفر بواسطة جين CLDN5 في الإنسان.[1][2][3]
الوظيفة
المراجع
- Sirotkin H، Morrow B، Saint-Jore B، Puech A، Das Gupta R، Patanjali SR، Skoultchi A، Weissman SM، Kucherlapati R (سبتمبر 1997). "Identification, characterization, and precise mapping of a human gene encoding a novel membrane-spanning protein from the 22q11 region deleted in velo-cardio-facial syndrome". Genomics. ج. 42 ع. 2: 245–51. DOI:10.1006/geno.1997.4734. PMID:9192844.
- Peacock RE، Keen TJ، Inglehearn CF (ديسمبر 1997). "Analysis of a human gene homologous to rat ventral prostate.1 protein". Genomics. ج. 46 ع. 3: 443–9. DOI:10.1006/geno.1997.5033. PMID:9441748.
- "Entrez Gene: CLDN5 claudin 5 (transmembrane protein deleted in velocardiofacial syndrome)". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Kniesel U، Wolburg H (2000). "Tight junctions of the blood-brain barrier". Cell. Mol. Neurobiol. ج. 20 ع. 1: 57–76. DOI:10.1023/A:1006995910836. PMID:10690502.
- Heiskala M، Peterson PA، Yang Y (2001). "The roles of claudin superfamily proteins in paracellular transport". Traffic. ج. 2 ع. 2: 93–8. DOI:10.1034/j.1600-0854.2001.020203.x. PMID:11247307.
- Tsukita S، Furuse M، Itoh M (2001). "Multifunctional strands in tight junctions". Nat. Rev. Mol. Cell Biol. ج. 2 ع. 4: 285–93. DOI:10.1038/35067088. PMID:11283726.
- Tsukita S، Furuse M (2002). "Claudin-based barrier in simple and stratified cellular sheets". Curr. Opin. Cell Biol. ج. 14 ع. 5: 531–6. DOI:10.1016/S0955-0674(02)00362-9. PMID:12231346.
- González-Mariscal L، Betanzos A، Nava P، Jaramillo BE (2003). "Tight junction proteins". Prog. Biophys. Mol. Biol. ج. 81 ع. 1: 1–44. DOI:10.1016/S0079-6107(02)00037-8. PMID:12475568.
- King JE، Eugenin EA، Buckner CM، Berman JW (2006). "HIV tat and neurotoxicity". Microbes Infect. ج. 8 ع. 5: 1347–57. DOI:10.1016/j.micinf.2005.11.014. PMID:16697675.
- Maruyama K، Sugano S (1994). "Oligo-capping: a simple method to replace the cap structure of eukaryotic mRNAs with oligoribonucleotides". Gene. ج. 138 ع. 1–2: 171–4. DOI:10.1016/0378-1119(94)90802-8. PMID:8125298.
- Suzuki Y، Yoshitomo-Nakagawa K، Maruyama K، Suyama A، Sugano S (1997). "Construction and characterization of a full length-enriched and a 5'-end-enriched cDNA library". Gene. ج. 200 ع. 1–2: 149–56. DOI:10.1016/S0378-1119(97)00411-3. PMID:9373149.
- Morita K، Furuse M، Fujimoto K، Tsukita S (1999). "Claudin multigene family encoding four-transmembrane domain protein components of tight junction strands". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 2: 511–6. DOI:10.1073/pnas.96.2.511. PMC:15167. PMID:9892664.
- Itoh M، Furuse M، Morita K، Kubota K، Saitou M، Tsukita S (1999). "Direct binding of three tight junction-associated MAGUKs, ZO-1, ZO-2, and ZO-3, with the COOH termini of claudins". J. Cell Biol. ج. 147 ع. 6: 1351–63. DOI:10.1083/jcb.147.6.1351. PMC:2168087. PMID:10601346.
- Kojima S، Rahner C، Peng S، Rizzolo LJ (2002). "Claudin 5 is transiently expressed during the development of the retinal pigment epithelium". J. Membr. Biol. ج. 186 ع. 2: 81–8. DOI:10.1007/s00232-001-0137-7. PMID:11944085.
- Poliak S، Matlis S، Ullmer C، Scherer SS، Peles E (2002). "Distinct claudins and associated PDZ proteins form different autotypic tight junctions in myelinating Schwann cells". J. Cell Biol. ج. 159 ع. 2: 361–72. DOI:10.1083/jcb.200207050. PMC:2173042. PMID:12403818.
- Helms HC، Waagepetersen HS، Nielsen CU، Brodin B (2010). "Paracellular tightness and claudin-5 expression is increased in the BCEC/astrocyte blood-brain barrier model by increasing media buffer capacity during growth". AAPS J. ج. 12 ع. 4: 759–70. DOI:10.1208/s12248-010-9237-6. PMC:2976989. PMID:20967520.
- Liu F، Koval M، Ranganathan S، Fanayan S، Hancock WS، Lundberg EK، Beavis RC، Lane L، Duek P، McQuade L، Kelleher NL، Baker MS (2015). "A systems proteomics view of the endogenous human claudin protein family". J Proteome Res. DOI:10.1021/acs.jproteome.5b00769. PMID:26680015.
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