ATF7IP
ATF7IP (Activating transcription factor 7 interacting protein) هوَ بروتين يُشَفر بواسطة جين ATF7IP في الإنسان.[1][2][3]
الوظيفة
المراجع
- De Graeve F، Bahr A، Chatton B، Kedinger C (يونيو 2000). "A murine ATFa-associated factor with transcriptional repressing activity". Oncogene. ج. 19 ع. 14: 1807–19. DOI:10.1038/sj.onc.1203492. PMID:10777215.
- Gunther M، Laithier M، Brison O (ديسمبر 2000). "A set of proteins interacting with transcription factor Sp1 identified in a two-hybrid screening". Mol Cell Biochem. ج. 210 ع. 1–2: 131–42. DOI:10.1023/A:1007177623283. PMID:10976766.
- "Entrez Gene: ATF7IP activating transcription factor 7 interacting protein". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. Bibcode:2002PNAS...9916899M. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Yamada K، Kawata H، Shou Z، وآخرون (2003). "Analysis of zinc-fingers and homeoboxes (ZHX)-1-interacting proteins: molecular cloning and characterization of a member of the ZHX family, ZHX3". Biochem. J. ج. 373 ع. Pt 1: 167–78. DOI:10.1042/BJ20021866. PMC:1223464. PMID:12659632.
- Fujita N، Watanabe S، Ichimura T، وآخرون (2003). "MCAF Mediates MBD1-Dependent Transcriptional Repression". Mol. Cell. Biol. ج. 23 ع. 8: 2834–43. DOI:10.1128/MCB.23.8.2834-2843.2003. PMC:152570. PMID:12665582.
- Morris JA، Kandpal G، Ma L، Austin CP (2004). "DISC1 (Disrupted-In-Schizophrenia 1) is a centrosome-associated protein that interacts with MAP1A, MIPT3, ATF4/5 and NUDEL: regulation and loss of interaction with mutation". Hum. Mol. Genet. ج. 12 ع. 13: 1591–608. DOI:10.1093/hmg/ddg162. PMID:12812986.
- Wang H، An W، Cao R، وآخرون (2003). "mAM facilitates conversion by ESET of dimethyl to trimethyl lysine 9 of histone H3 to cause transcriptional repression". Mol. Cell. ج. 12 ع. 2: 475–87. DOI:10.1016/j.molcel.2003.08.007. PMID:14536086.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Colland F، Jacq X، Trouplin V، وآخرون (2004). "Functional Proteomics Mapping of a Human Signaling Pathway". Genome Res. ج. 14 ع. 7: 1324–32. DOI:10.1101/gr.2334104. PMC:442148. PMID:15231748.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The Status, Quality, and Expansion of the NIH Full-Length cDNA Project: The Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Ichimura T، Watanabe S، Sakamoto Y، وآخرون (2005). "Transcriptional repression and heterochromatin formation by MBD1 and MCAF/AM family proteins". J. Biol. Chem. ج. 280 ع. 14: 13928–35. DOI:10.1074/jbc.M413654200. PMID:15691849.
- Chang LK، Chung JY، Hong YR، وآخرون (2005). "Activation of Sp1-mediated transcription by Rta of Epstein–Barr virus via an interaction with MCAF1". Nucleic Acids Res. ج. 33 ع. 20: 6528–39. DOI:10.1093/nar/gki956. PMC:1298921. PMID:16314315.
- Uchimura Y، Ichimura T، Uwada J، وآخرون (2006). "Involvement of SUMO modification in MBD1- and MCAF1-mediated heterochromatin formation". J. Biol. Chem. ج. 281 ع. 32: 23180–90. DOI:10.1074/jbc.M602280200. PMID:16757475.
- Olsen JV، Blagoev B، Gnad F، وآخرون (2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. ج. 127 ع. 3: 635–48. DOI:10.1016/j.cell.2006.09.026. PMID:17081983.
- بوابة الكيمياء الحيوية
- بوابة طب
- بوابة علم الأحياء الخلوي والجزيئي
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