ATF7
ATF7 (Activating transcription factor 7) هوَ بروتين يُشَفر بواسطة جين ATF7 في الإنسان.[1][2][3]
الوظيفة
المراجع
- Peters CS، Liang X، Li S، Kannan S، Peng Y، Taub R، Diamond RH (أبريل 2001). "ATF-7, a novel bZIP protein, interacts with the PRL-1 protein-tyrosine phosphatase". J Biol Chem. ج. 276 ع. 17: 13718–26. DOI:10.1074/jbc.M011562200. PMID:11278933.
- "Entrez Gene: ATF7 activating transcription factor 7". مؤرشف من الأصل في 2010-12-05.
- Gaire M، Chatton B، Kedinger C (أغسطس 1990). "Isolation and characterization of two novel, closely related ATF cDNA clones from HeLa cells". Nucleic Acids Res. ج. 18 ع. 12: 3467–73. DOI:10.1093/nar/18.12.3467. PMC:330998. PMID:1694576.
قراءة متعمقة
- Olsen JV، Blagoev B، Gnad F، Macek B، Kumar C، Mortensen P، Mann M (2006). "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks". Cell. ج. 127 ع. 3: 635–48. DOI:10.1016/j.cell.2006.09.026. PMID:17081983.
- Hamard PJ، Dalbies-Tran R، Hauss C، Davidson I، Kedinger C، Chatton B (2005). "A functional interaction between ATF7 and TAF12 that is modulated by TAF4". Oncogene. ج. 24 ع. 21: 3472–83. DOI:10.1038/sj.onc.1208565. PMID:15735663.
- Hamard PJ، Boyer-Guittaut M، Camuzeaux B، Dujardin D، Hauss C، Oelgeschläger T، Vigneron M، Kedinger C، Chatton B (2007). "Sumoylation delays the ATF7 transcription factor subcellular localization and inhibits its transcriptional activity". Nucleic Acids Res. ج. 35 ع. 4: 1134–44. DOI:10.1093/nar/gkl1168. PMC:1851647. PMID:17264123.
- Breitwieser W، Lyons S، Flenniken AM، Ashton G، Bruder G، Willington M، Lacaud G، Kouskoff V، Jones N (2007). "Feedback regulation of p38 activity via ATF2 is essential for survival of embryonic liver cells". Genes Dev. ج. 21 ع. 16: 2069–82. DOI:10.1101/gad.430207. PMC:1948861. PMID:17699753.
- Newman JR، Keating AE (2003). "Comprehensive identification of human bZIP interactions with coiled-coil arrays". Science. ج. 300 ع. 5628: 2097–101. DOI:10.1126/science.1084648. PMID:12805554.
- Newman SP، Bates NP، Vernimmen D، Parker MG، Hurst HC (2000). "Cofactor competition between the ligand-bound oestrogen receptor and an intron 1 enhancer leads to oestrogen repression of ERBB2 expression in breast cancer". Oncogene. ج. 19 ع. 4: 490–7. DOI:10.1038/sj.onc.1203416. PMID:10698518.
- Goetz J، Chatton B، Mattei MG، Kedinger C (1997). "Structure and expression of the ATFa gene". J. Biol. Chem. ج. 271 ع. 47: 29589–98. DOI:10.1074/jbc.271.47.29589. PMID:8939888.
- Bocco JL، Bahr A، Goetz J، Hauss C، Kallunki T، Kedinger C، Chatton B (1996). "In vivo association of ATFa with JNK/SAP kinase activities". Oncogene. ج. 12 ع. 9: 1971–80. PMID:8649858.
- Pescini R، Kaszubska W، Whelan J، DeLamarter JF، Hooft van Huijsduijnen R (1994). "ATF-a0, a novel variant of the ATF/CREB transcription factor family, forms a dominant transcription inhibitor in ATF-a heterodimers". J. Biol. Chem. ج. 269 ع. 2: 1159–65. PMID:8288576.
- Zhou Q، Gedrich RW، Engel DA (1995). "Transcriptional repression of the c-fos gene by YY1 is mediated by a direct interaction with ATF/CREB". J. Virol. ج. 69 ع. 7: 4323–30. PMC:189172. PMID:7769693.
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة طب
- بوابة الكيمياء الحيوية
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