مستقبل ريتينويد أكس ألفا

مستقبل ريتينويد أكس ألفا
المعرفات
الرمز	RXRA
Ensembl Transcript ID	ENST00000484822; ENST00000356384; ENST00000481739; ENST00000649020; ENST00000672570
Ensembl Gene ID	ENSG00000186350
أنتريه	6256
HUGO	10477
أوميم	180245
هومولوجين	2220
RefSeq	NM_002957
يونيبروت	P19793
بيانات أخرى
الموقع الكروموسومي	Chr. 9 q34

Retinoid X receptor, alpha
	بنك بيانات البروتينات rendering based on 1by4.
التراكيب المتوفرة
بنك بيانات البروتين	بحث أورثولوغ: PDBe, RCSB
قائمة رموز معرفات بنك بيانات البروتين
	1BY4, 1DSZ, 1FBY, 1FM6, 1FM9, 1G1U, 1G5Y, 1K74, 1LBD, 1MV9, 1MVC, 1MZN, 1R0N, 1RDT, 1RXR, 1XLS, 1XV9, 1XVP, 1YNW, 2ACL, 2NLL, 2P1T, 2P1U, 2P1V, 2ZXZ, 2ZY0, 3DZU, 3DZY, 3E00, 3E94, 3FAL, 3FC6, 3FUG, 3H0A, 3KWY, 3NSP, 3NSQ, 3OAP, 3OZJ, 3PCU, 3R29, 3R2A, 3R5M, 3UVV, 4CN2, 4CN3, 4CN5, 4CN7, 4J5W, 4J5X, 4K4J, 4K6I, 4M8E, 4M8H, 4N5G, 4N8R, 4NQA, 4OC7, 4POH, 4POJ, 4PP3, 4PP5
المعرفات
الرمز، (أو الرموز)	RXRA; NR2B1
معرفات خارجية	OMIM: 180245 MGI: 98214 هومولوجين: 2220 الاتحاد الدولي للصيدلة الأساسية والسريرية: 610 مختبر علم الأحياء الجزيئي الأوروبي الكيميائي: 2061 بطاقات الجينات: RXRA Gene
علم الوجود الجيني
الوظيفة الجزيئية	• RNA polymerase II regulatory region sequence-specific DNA binding; • DNA binding; • double-stranded DNA binding; • transcription factor activity, sequence-specific DNA binding; • steroid hormone receptor activity; • retinoic acid receptor activity; • transcription coactivator activity; • RNA polymerase II transcription factor activity, ligand-activated sequence-specific DNA binding; • 9-cis retinoic acid receptor activity; • protein binding; • zinc ion binding; • enzyme binding; • protein domain specific binding; • chromatin DNA binding; • protein complex binding; • vitamin D receptor binding; • retinoic acid receptor binding; • sequence-specific DNA binding; • transcription regulatory region DNA binding; • retinoic acid-responsive element binding; • protein heterodimerization activity; • vitamin D response element binding;
المكونات الخلوية	• nuclear chromatin; • nucleus; • nucleoplasm; • axon; • receptor complex; • RNA polymerase II transcription factor complex;
العملية الإحيائية	• negative regulation of transcription from RNA polymerase II promoter; • in utero embryonic development; • liver development; • maternal placenta development; • transcription initiation from RNA polymerase II promoter; • vitamin metabolic process; • inflammatory response; • midgut development; • embryo implantation; • aging; • cholesterol metabolic process; • bile acid metabolic process; • negative regulation of cell proliferation; • response to selenium ion; • gene expression; • bile acid and bile salt transport; • modulation by virus of host morphology or physiology; • axon regeneration; • regulation of myelination; • response to retinoic acid; • cellular response to insulin stimulus; • response to vitamin A; • response to vitamin D; • peroxisome proliferator activated receptor signaling pathway; • camera-type eye development; • positive regulation of apoptotic process; • steroid hormone mediated signaling pathway; • cellular lipid metabolic process; • small molecule metabolic process; • response to ethanol; • positive regulation of transcription from RNA polymerase II promoter; • positive regulation of translational initiation by iron; • retinoic acid receptor signaling pathway; • protein homotetramerization; • response to glucocorticoid; • ventricular cardiac muscle tissue morphogenesis; • ventricular cardiac muscle cell differentiation; • secretory columnal luminar epithelial cell differentiation involved in prostate glandular acinus development; • regulation of branching involved in prostate gland morphogenesis; • positive regulation of transcription from RNA polymerase II promoter involved in cellular response to chemical stimulus;
	المصادر: Amigo / QuickGO
نمط التعبير عن الحمض النووي الريبوزي
	مصادر أخرى عن بيانات أنماط التعبير
التقويمات

مستقبل ريتينويد أكس ألفا (بالإنجليزية: Retinoid X receptor alpha)‏ (مختصره RXR-alpha), ويعرف كذلك مستقبل نووي عائلة 2، مجموعة ب، عضو 1 (بالإنجليزية: nuclear receptor subfamily 2, group B, member 1)‏ (مختصره NR2B1) هو is a مستقبل نووي موجود عند الإنسان مشفر في المورثة RXRA.[1]

مخطط مسار تفاعلي

اضغط على المورثة أو البروتين أو المادة للذهاب إلى المقالة المتعلقة بها.[§ 1]

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|alt=مسار تكوين الفيتامين د (عرض / تعديل)]]

مسار تكوين الفيتامين د (عرض / تعديل)

يمكن تعديل مخطط مسار التكوين التفاعلي في ويكيباثويز: "VitaminDSynthesis_WP1531".

التفاعلات

أظهر مستقبل ريتنويد أكس ألفا تآثرات مع:

BCL3,[2]
BRD8,[3]
CLOCK,[4]
FXR[5]
IGFBP3,[6]
ITGB3BP,[7]
LXR-β,[5]
MyoD,[8]
NCOA6,[9][10][11][12]
NFKBIB,[13]
NPAS2,[4]
NRIP1,[14][15]
NR4A1,[16]
NCOA2,[17]
NCOA3,[18]
POU2F1,[19][20]
PPARGC1A,[21]
مستقبل منشط لمكاثر البيروكسيسوم النوع-غاما,[22][23][24]
RNF8,[25]
مستقبل حمض الريتينويك ألفا,[26][27]
SHP,[28][29]
TADA3L,[30]
TBP,[31]
TRIM24,[32][33]
TR-β,[34][35]
مستقبل كالسيتريول.[17][36]

طالع أيضا

مستقبل ريتنويد أكس

المصادر

Mangelsdorf DJ، Ong ES، Dyck JA، Evans RM (مايو 1990). "Nuclear receptor that identifies a novel retinoic acid response pathway". Nature. ج. 345 ع. 6272: 224–9. Bibcode:1990Natur.345..224M. DOI:10.1038/345224a0. PMID:2159111.
Na SY، Choi HS، Kim JW، Na DS، Lee JW (1998). "Bcl3, an IkappaB protein, as a novel transcription coactivator of the retinoid X receptor". J. Biol. Chem. ج. 273 ع. 47: 30933–8. DOI:10.1074/jbc.273.47.30933. PMID:9812988.
Monden T، Kishi M، Hosoya T، Satoh T، Wondisford FE، Hollenberg AN، Yamada M، Mori M (1999). "p120 acts as a specific coactivator for 9-cis-retinoic acid receptor (RXR) on peroxisome proliferator-activated receptor-gamma/RXR heterodimers". Mol. Endocrinol. ج. 13 ع. 10: 1695–703. DOI:10.1210/me.13.10.1695. PMID:10517671.
McNamara P، Seo SB، Rudic RD، Sehgal A، Chakravarti D، FitzGerald GA (2001). "Regulation of CLOCK and MOP4 by nuclear hormone receptors in the vasculature: a humoral mechanism to reset a peripheral clock". Cell. ج. 105 ع. 7: 877–89. DOI:10.1016/S0092-8674(01)00401-9. PMID:11439184.
Seol W، Choi HS، Moore DD (1995). "Isolation of proteins that interact specifically with the retinoid X receptor: two novel orphan receptors". Mol. Endocrinol. ج. 9 ع. 1: 72–85. DOI:10.1210/mend.9.1.7760852. PMID:7760852.
Liu B، Lee HY، Weinzimer SA، Powell DR، Clifford JL، Kurie JM، Cohen P (2000). "Direct functional interactions between insulin-like growth factor-binding protein-3 and retinoid X receptor-alpha regulate transcriptional signaling and apoptosis". J. Biol. Chem. ج. 275 ع. 43: 33607–13. DOI:10.1074/jbc.M002547200. PMID:10874028.
Li D، Desai-Yajnik V، Lo E، Schapira M، Abagyan R، Samuels HH (1999). "NRIF3 is a novel coactivator mediating functional specificity of nuclear hormone receptors". Mol. Cell. Biol. ج. 19 ع. 10: 7191–202. PMC:84712. PMID:10490654.
Froeschlé A، Alric S، Kitzmann M، Carnac G، Auradé F، Rochette-Egly C، Bonnieu A (1998). "Retinoic acid receptors and muscle b-HLH proteins: partners in retinoid-induced myogenesis". Oncogene. ج. 16 ع. 26: 3369–78. DOI:10.1038/sj.onc.1201894. PMID:9692544.
Lee SK، Anzick SL، Choi JE، Bubendorf L، Guan XY، Jung YK، Kallioniemi OP، Kononen J، Trent JM، Azorsa D، Jhun BH، Cheong JH، Lee YC، Meltzer PS، Lee JW (1999). "A nuclear factor, ASC-2, as a cancer-amplified transcriptional coactivator essential for ligand-dependent transactivation by nuclear receptors in vivo". J. Biol. Chem. ج. 274 ع. 48: 34283–93. DOI:10.1074/jbc.274.48.34283. PMID:10567404.
Lee SK، Jung SY، Kim YS، Na SY، Lee YC، Lee JW (2001). "Two distinct nuclear receptor-interaction domains and CREB-binding protein-dependent transactivation function of activating signal cointegrator-2". Mol. Endocrinol. ج. 15 ع. 2: 241–54. DOI:10.1210/me.15.2.241. PMID:11158331.
Kong HJ، Park MJ، Hong S، Yu HJ، Lee YC، Choi YH، Cheong J (2003). "Hepatitis B virus X protein regulates transactivation activity and protein stability of the cancer-amplified transcription coactivator ASC-2". Hepatology. ج. 38 ع. 5: 1258–66. DOI:10.1053/jhep.2003.50451. PMID:14578865.
Ko L، Cardona GR، Iwasaki T، Bramlett KS، Burris TP، Chin WW (2002). "Ser-884 adjacent to the LXXLL motif of coactivator TRBP defines selectivity for ERs and TRs". Mol. Endocrinol. ج. 16 ع. 1: 128–40. DOI:10.1210/mend.16.1.0755. PMID:11773444.
Na SY، Kim HJ، Lee SK، Choi HS، Na DS، Lee MO، Chung M، Moore DD، Lee JW (1998). "IkappaBbeta interacts with the retinoid X receptor and inhibits retinoid-dependent transactivation in lipopolysaccharide-treated cells". J. Biol. Chem. ج. 273 ع. 6: 3212–5. DOI:10.1074/jbc.273.6.3212. PMID:9452433.
Farooqui M، Franco PJ، Thompson J، Kagechika H، Chandraratna RA، Banaszak L، Wei LN (2003). "Effects of retinoid ligands on RIP140: molecular interaction with retinoid receptors and biological activity". Biochemistry. ج. 42 ع. 4: 971–9. DOI:10.1021/bi020497k. PMID:12549917.
L'Horset F، Dauvois S، Heery DM، Cavaillès V، Parker MG (1996). "RIP-140 interacts with multiple nuclear receptors by means of two distinct sites". Mol. Cell. Biol. ج. 16 ع. 11: 6029–36. PMC:231605. PMID:8887632.
Lin B، Kolluri SK، Lin F، Liu W، Han YH، Cao X، Dawson MI، Reed JC، Zhang XK (2004). "Conversion of Bcl-2 from protector to killer by interaction with nuclear orphan receptor Nur77/TR3". Cell. ج. 116 ع. 4: 527–40. DOI:10.1016/S0092-8674(04)00162-X. PMID:14980220.
Zhang C، Baudino TA، Dowd DR، Tokumaru H، Wang W، MacDonald PN (2001). "Ternary complexes and cooperative interplay between NCoA-62/Ski-interacting protein and steroid receptor coactivators in vitamin D receptor-mediated transcription". J. Biol. Chem. ج. 276 ع. 44: 40614–20. DOI:10.1074/jbc.M106263200. PMID:11514567.
Chen H، Lin RJ، Schiltz RL، Chakravarti D، Nash A، Nagy L، Privalsky ML، Nakatani Y، Evans RM (1997). "Nuclear receptor coactivator ACTR is a novel histone acetyltransferase and forms a multimeric activation complex with P/CAF and CBP/p300". Cell. ج. 90 ع. 3: 569–80. DOI:10.1016/S0092-8674(00)80516-4. PMID:9267036.
Préfontaine GG، Walther R، Giffin W، Lemieux ME، Pope L، Haché RJ (1999). "Selective binding of steroid hormone receptors to octamer transcription factors determines transcriptional synergism at the mouse mammary tumor virus promoter". J. Biol. Chem. ج. 274 ع. 38: 26713–9. DOI:10.1074/jbc.274.38.26713. PMID:10480874.
Kakizawa T، Miyamoto T، Ichikawa K، Kaneko A، Suzuki S، Hara M، Nagasawa T، Takeda T، Mori Ji، Kumagai M، Hashizume K (1999). "Functional interaction between Oct-1 and retinoid X receptor". J. Biol. Chem. ج. 274 ع. 27: 19103–8. DOI:10.1074/jbc.274.27.19103. PMID:10383413.
Delerive P، Wu Y، Burris TP، Chin WW، Suen CS (2002). "PGC-1 functions as a transcriptional coactivator for the retinoid X receptors". J. Biol. Chem. ج. 277 ع. 6: 3913–7. DOI:10.1074/jbc.M109409200. PMID:11714715.
Tontonoz P، Graves RA، Budavari AI، Erdjument-Bromage H، Lui M، Hu E، Tempst P، Spiegelman BM (1994). "Adipocyte-specific transcription factor ARF6 is a heterodimeric complex of two nuclear hormone receptors, PPAR gamma and RXR alpha". Nucleic Acids Res. ج. 22 ع. 25: 5628–34. DOI:10.1093/nar/22.25.5628. PMC:310126. PMID:7838715.
Berger J، Patel HV، Woods J، Hayes NS، Parent SA، Clemas J، Leibowitz MD، Elbrecht A، Rachubinski RA، Capone JP، Moller DE (2000). "A PPARgamma mutant serves as a dominant negative inhibitor of PPAR signaling and is localized in the nucleus". Mol. Cell. Endocrinol. ج. 162 ع. 1–2: 57–67. DOI:10.1016/S0303-7207(00)00211-2. PMID:10854698.
Gampe RT، Montana VG، Lambert MH، Miller AB، Bledsoe RK، Milburn MV، Kliewer SA، Willson TM، Xu HE (2000). "Asymmetry in the PPARgamma/RXRalpha crystal structure reveals the molecular basis of heterodimerization among nuclear receptors". Mol. Cell. ج. 5 ع. 3: 545–55. DOI:10.1016/S1097-2765(00)80448-7. PMID:10882139.
Takano Y، Adachi S، Okuno M، Muto Y، Yoshioka T، Matsushima-Nishiwaki R، Tsurumi H، Ito K، Friedman SL، Moriwaki H، Kojima S، Okano Y (2004). "The RING finger protein, RNF8, interacts with retinoid X receptor alpha and enhances its transcription-stimulating activity". J. Biol. Chem. ج. 279 ع. 18: 18926–34. DOI:10.1074/jbc.M309148200. PMID:14981089.
Benkoussa M، Brand C، Delmotte MH، Formstecher P، Lefebvre P (2002). "Retinoic acid receptors inhibit AP1 activation by regulating extracellular signal-regulated kinase and CBP recruitment to an AP1-responsive promoter". Mol. Cell. Biol. ج. 22 ع. 13: 4522–34. DOI:10.1128/MCB.22.13.4522-4534.2002. PMC:133906. PMID:12052862.
Bugge TH، Pohl J، Lonnoy O، Stunnenberg HG (1992). "RXR alpha, a promiscuous partner of retinoic acid and thyroid hormone receptors". EMBO J. ج. 11 ع. 4: 1409–18. PMC:556590. PMID:1314167.
Lee YK، Dell H، Dowhan DH، Hadzopoulou-Cladaras M، Moore DD (2000). "The orphan nuclear receptor SHP inhibits hepatocyte nuclear factor 4 and retinoid X receptor transactivation: two mechanisms for repression". Mol. Cell. Biol. ج. 20 ع. 1: 187–95. DOI:10.1128/MCB.20.1.187-195.2000. PMC:85074. PMID:10594021.
Brendel C، Schoonjans K، Botrugno OA، Treuter E، Auwerx J (2002). "The small heterodimer partner interacts with the liver X receptor alpha and represses its transcriptional activity". Mol. Endocrinol. ج. 16 ع. 9: 2065–76. DOI:10.1210/me.2001-0194. PMID:12198243.
Zeng M، Kumar A، Meng G، Gao Q، Dimri G، Wazer D، Band H، Band V (2002). "Human papilloma virus 16 E6 oncoprotein inhibits retinoic X receptor-mediated transactivation by targeting human ADA3 coactivator". J. Biol. Chem. ج. 277 ع. 47: 45611–8. DOI:10.1074/jbc.M208447200. PMID:12235159.
Schulman IG، Chakravarti D، Juguilon H، Romo A، Evans RM (1995). "Interactions between the retinoid X receptor and a conserved region of the TATA-binding protein mediate hormone-dependent transactivation". Proc. Natl. Acad. Sci. U.S.A. ج. 92 ع. 18: 8288–92. Bibcode:1995PNAS...92.8288S. DOI:10.1073/pnas.92.18.8288. PMC:41142. PMID:7667283.
Thénot S، Henriquet C، Rochefort H، Cavaillès V (1997). "Differential interaction of nuclear receptors with the putative human transcriptional coactivator hTIF1". J. Biol. Chem. ج. 272 ع. 18: 12062–8. DOI:10.1074/jbc.272.18.12062. PMID:9115274.
Lee WY، Noy N (2002). "Interactions of RXR with coactivators are differentially mediated by helix 11 of the receptor's ligand binding domain". Biochemistry. ج. 41 ع. 8: 2500–8. DOI:10.1021/bi011764. PMID:11851396.
Monden T، Wondisford FE، Hollenberg AN (1997). "Isolation and characterization of a novel ligand-dependent thyroid hormone receptor-coactivating protein". J. Biol. Chem. ج. 272 ع. 47: 29834–41. DOI:10.1074/jbc.272.47.29834. PMID:9368056.
Jeyakumar M، Tanen MR، Bagchi MK (1997). "Analysis of the functional role of steroid receptor coactivator-1 in ligand-induced transactivation by thyroid hormone receptor". Mol. Endocrinol. ج. 11 ع. 6: 755–67. DOI:10.1210/mend.11.6.0003. PMID:9171239.
Baudino TA، Kraichely DM، Jefcoat SC، Winchester SK، Partridge NC، MacDonald PN (1998). "Isolation and characterization of a novel coactivator protein, NCoA-62, involved in vitamin D-mediated transcription". J. Biol. Chem. ج. 273 ع. 26: 16434–41. DOI:10.1074/jbc.273.26.16434. PMID:9632709.

بوابة علم الأحياء
بوابة الكيمياء الحيوية
بوابة علم الأحياء الخلوي والجزيئي

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[WikiPathways-2] يمكن تعديل مخطط مسار التكوين التفاعلي في ويكيباثويز: "VitaminDSynthesis_WP1531".

[pmid2159111-1] Mangelsdorf DJ، Ong ES، Dyck JA، Evans RM (مايو 1990). "Nuclear receptor that identifies a novel retinoic acid response pathway". Nature. ج. 345 ع. 6272: 224–9. Bibcode:1990Natur.345..224M. DOI:10.1038/345224a0. PMID:2159111.

[pmid9812988-3] Na SY، Choi HS، Kim JW، Na DS، Lee JW (1998). "Bcl3, an IkappaB protein, as a novel transcription coactivator of the retinoid X receptor". J. Biol. Chem. ج. 273 ع. 47: 30933–8. DOI:10.1074/jbc.273.47.30933. PMID:9812988.

[pmid10517671-4] Monden T، Kishi M، Hosoya T، Satoh T، Wondisford FE، Hollenberg AN، Yamada M، Mori M (1999). "p120 acts as a specific coactivator for 9-cis-retinoic acid receptor (RXR) on peroxisome proliferator-activated receptor-gamma/RXR heterodimers". Mol. Endocrinol. ج. 13 ع. 10: 1695–703. DOI:10.1210/me.13.10.1695. PMID:10517671.

[pmid11439184-5] McNamara P، Seo SB، Rudic RD، Sehgal A، Chakravarti D، FitzGerald GA (2001). "Regulation of CLOCK and MOP4 by nuclear hormone receptors in the vasculature: a humoral mechanism to reset a peripheral clock". Cell. ج. 105 ع. 7: 877–89. DOI:10.1016/S0092-8674(01)00401-9. PMID:11439184.

[pmid7760852-6] Seol W، Choi HS، Moore DD (1995). "Isolation of proteins that interact specifically with the retinoid X receptor: two novel orphan receptors". Mol. Endocrinol. ج. 9 ع. 1: 72–85. DOI:10.1210/mend.9.1.7760852. PMID:7760852.

[pmid10874028-7] Liu B، Lee HY، Weinzimer SA، Powell DR، Clifford JL، Kurie JM، Cohen P (2000). "Direct functional interactions between insulin-like growth factor-binding protein-3 and retinoid X receptor-alpha regulate transcriptional signaling and apoptosis". J. Biol. Chem. ج. 275 ع. 43: 33607–13. DOI:10.1074/jbc.M002547200. PMID:10874028.

[pmid10490654-8] Li D، Desai-Yajnik V، Lo E، Schapira M، Abagyan R، Samuels HH (1999). "NRIF3 is a novel coactivator mediating functional specificity of nuclear hormone receptors". Mol. Cell. Biol. ج. 19 ع. 10: 7191–202. PMC:84712. PMID:10490654.

[pmid9692544-9] Froeschlé A، Alric S، Kitzmann M، Carnac G، Auradé F، Rochette-Egly C، Bonnieu A (1998). "Retinoic acid receptors and muscle b-HLH proteins: partners in retinoid-induced myogenesis". Oncogene. ج. 16 ع. 26: 3369–78. DOI:10.1038/sj.onc.1201894. PMID:9692544.

[pmid10567404-10] Lee SK، Anzick SL، Choi JE، Bubendorf L، Guan XY، Jung YK، Kallioniemi OP، Kononen J، Trent JM، Azorsa D، Jhun BH، Cheong JH، Lee YC، Meltzer PS، Lee JW (1999). "A nuclear factor, ASC-2, as a cancer-amplified transcriptional coactivator essential for ligand-dependent transactivation by nuclear receptors in vivo". J. Biol. Chem. ج. 274 ع. 48: 34283–93. DOI:10.1074/jbc.274.48.34283. PMID:10567404.

[pmid11158331-11] Lee SK، Jung SY، Kim YS، Na SY، Lee YC، Lee JW (2001). "Two distinct nuclear receptor-interaction domains and CREB-binding protein-dependent transactivation function of activating signal cointegrator-2". Mol. Endocrinol. ج. 15 ع. 2: 241–54. DOI:10.1210/me.15.2.241. PMID:11158331.

[pmid14578865-12] Kong HJ، Park MJ، Hong S، Yu HJ، Lee YC، Choi YH، Cheong J (2003). "Hepatitis B virus X protein regulates transactivation activity and protein stability of the cancer-amplified transcription coactivator ASC-2". Hepatology. ج. 38 ع. 5: 1258–66. DOI:10.1053/jhep.2003.50451. PMID:14578865.

[pmid11773444-13] Ko L، Cardona GR، Iwasaki T، Bramlett KS، Burris TP، Chin WW (2002). "Ser-884 adjacent to the LXXLL motif of coactivator TRBP defines selectivity for ERs and TRs". Mol. Endocrinol. ج. 16 ع. 1: 128–40. DOI:10.1210/mend.16.1.0755. PMID:11773444.

[pmid9452433-14] Na SY، Kim HJ، Lee SK، Choi HS، Na DS، Lee MO، Chung M، Moore DD، Lee JW (1998). "IkappaBbeta interacts with the retinoid X receptor and inhibits retinoid-dependent transactivation in lipopolysaccharide-treated cells". J. Biol. Chem. ج. 273 ع. 6: 3212–5. DOI:10.1074/jbc.273.6.3212. PMID:9452433.

[pmid12549917-15] Farooqui M، Franco PJ، Thompson J، Kagechika H، Chandraratna RA، Banaszak L، Wei LN (2003). "Effects of retinoid ligands on RIP140: molecular interaction with retinoid receptors and biological activity". Biochemistry. ج. 42 ع. 4: 971–9. DOI:10.1021/bi020497k. PMID:12549917.

[pmid8887632-16] L'Horset F، Dauvois S، Heery DM، Cavaillès V، Parker MG (1996). "RIP-140 interacts with multiple nuclear receptors by means of two distinct sites". Mol. Cell. Biol. ج. 16 ع. 11: 6029–36. PMC:231605. PMID:8887632.

[pmid14980220-17] Lin B، Kolluri SK، Lin F، Liu W، Han YH، Cao X، Dawson MI، Reed JC، Zhang XK (2004). "Conversion of Bcl-2 from protector to killer by interaction with nuclear orphan receptor Nur77/TR3". Cell. ج. 116 ع. 4: 527–40. DOI:10.1016/S0092-8674(04)00162-X. PMID:14980220.

[pmid11514567-18] Zhang C، Baudino TA، Dowd DR، Tokumaru H، Wang W، MacDonald PN (2001). "Ternary complexes and cooperative interplay between NCoA-62/Ski-interacting protein and steroid receptor coactivators in vitamin D receptor-mediated transcription". J. Biol. Chem. ج. 276 ع. 44: 40614–20. DOI:10.1074/jbc.M106263200. PMID:11514567.

[pmid9267036-19] Chen H، Lin RJ، Schiltz RL، Chakravarti D، Nash A، Nagy L، Privalsky ML، Nakatani Y، Evans RM (1997). "Nuclear receptor coactivator ACTR is a novel histone acetyltransferase and forms a multimeric activation complex with P/CAF and CBP/p300". Cell. ج. 90 ع. 3: 569–80. DOI:10.1016/S0092-8674(00)80516-4. PMID:9267036.

[pmid10480874-20] Préfontaine GG، Walther R، Giffin W، Lemieux ME، Pope L، Haché RJ (1999). "Selective binding of steroid hormone receptors to octamer transcription factors determines transcriptional synergism at the mouse mammary tumor virus promoter". J. Biol. Chem. ج. 274 ع. 38: 26713–9. DOI:10.1074/jbc.274.38.26713. PMID:10480874.

[pmid10383413-21] Kakizawa T، Miyamoto T، Ichikawa K، Kaneko A، Suzuki S، Hara M، Nagasawa T، Takeda T، Mori Ji، Kumagai M، Hashizume K (1999). "Functional interaction between Oct-1 and retinoid X receptor". J. Biol. Chem. ج. 274 ع. 27: 19103–8. DOI:10.1074/jbc.274.27.19103. PMID:10383413.

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التصنيفات الطبية	دليل الوراثة المندلية عند البشر (OMIM): 180245
المعرفات الخارجية	Ensembl Transcript ID: ENST00000672570 نظام اللغة الطبية الموحدة (UMLS CUI): C1456392